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smad8  (Santa Cruz Biotechnology)


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    Santa Cruz Biotechnology smad8
    Figure 2. Differential expression of SMAD1–8 proteins in the peripheral facial nerve following injury. Western blot analysis of SMAD1–8 protein levels in the peripheral facial nerve at 2 and 12 weeks post injury induced by compression or axotomy. At 2 weeks, compression injury led to a significant increase in SMAD2 expression. By 12 weeks, SMAD7 and <t>SMAD8</t> expression were elevated in the Compression group. In contrast, axotomy resulted in significant upregulation of SMAD1, SMAD2, SMAD3, SMAD4, SMAD6, SMAD7, and SMAD8 at both 2 and 12 weeks; no change in SMAD5 expression was observed in either injury model. The complete gel images are available in Supplementary Figures S1 and S2 for reference. Data are presented as means ± S.D., with a sample size of n = 3 per group (* p < 0.05 vs. Sham group). Abbreviations: wks, weeks.
    Smad8, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 37 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/smad8/Smad8+Antibody/pm40076910-176-62-63
    Average 93 stars, based on 37 article reviews
    smad8 - by Bioz Stars, 2026-09
    93/100 stars

    Images

    1) Product Images from "Expression Patterns of SMAD1-8 in the Peripheral Facial Nerve Following Compressive Nerve Injury or Axotomy."

    Article Title: Expression Patterns of SMAD1-8 in the Peripheral Facial Nerve Following Compressive Nerve Injury or Axotomy.

    Journal: International journal of molecular sciences

    doi: 10.3390/ijms26052291

    Figure 2. Differential expression of SMAD1–8 proteins in the peripheral facial nerve following injury. Western blot analysis of SMAD1–8 protein levels in the peripheral facial nerve at 2 and 12 weeks post injury induced by compression or axotomy. At 2 weeks, compression injury led to a significant increase in SMAD2 expression. By 12 weeks, SMAD7 and SMAD8 expression were elevated in the Compression group. In contrast, axotomy resulted in significant upregulation of SMAD1, SMAD2, SMAD3, SMAD4, SMAD6, SMAD7, and SMAD8 at both 2 and 12 weeks; no change in SMAD5 expression was observed in either injury model. The complete gel images are available in Supplementary Figures S1 and S2 for reference. Data are presented as means ± S.D., with a sample size of n = 3 per group (* p < 0.05 vs. Sham group). Abbreviations: wks, weeks.
    Figure Legend Snippet: Figure 2. Differential expression of SMAD1–8 proteins in the peripheral facial nerve following injury. Western blot analysis of SMAD1–8 protein levels in the peripheral facial nerve at 2 and 12 weeks post injury induced by compression or axotomy. At 2 weeks, compression injury led to a significant increase in SMAD2 expression. By 12 weeks, SMAD7 and SMAD8 expression were elevated in the Compression group. In contrast, axotomy resulted in significant upregulation of SMAD1, SMAD2, SMAD3, SMAD4, SMAD6, SMAD7, and SMAD8 at both 2 and 12 weeks; no change in SMAD5 expression was observed in either injury model. The complete gel images are available in Supplementary Figures S1 and S2 for reference. Data are presented as means ± S.D., with a sample size of n = 3 per group (* p < 0.05 vs. Sham group). Abbreviations: wks, weeks.

    Techniques Used: Quantitative Proteomics, Western Blot, Expressing

    Related Articles

    Incubation:

    Article Title: BMP-7 attenuated silica-induced pulmonary fibrosis through modulation of the balance between TGF-β/Smad and BMP-7/Smad signaling pathway.
    Article Snippet: Objective: To investigate the anti-fibrotic effects and possible mechanisms of bone morphogenetic protein-7 (BMP-7) on silica induced fibrosis in RLE-6TN cells, and compare the preventive treatment of experimental silicosis with BMP-7 with therapeutic treatment of silicosis in vitro models.. Methods: RLE-6TN cells were incubated with the supernatant of RAW264.7, treated by 50 mg/mL silica in either presence or absence of BMP-7 in different phases.. Morphological changes and the cellular woundhealing assays were used to evaluate the process of EMT.



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    Figure 2. Differential expression of SMAD1–8 proteins in the peripheral facial nerve following injury. Western blot analysis of SMAD1–8 protein levels in the peripheral facial nerve at 2 and 12 weeks post injury induced by compression or axotomy. At 2 weeks, compression injury led to a significant increase in SMAD2 expression. By 12 weeks, SMAD7 and <t>SMAD8</t> expression were elevated in the Compression group. In contrast, axotomy resulted in significant upregulation of SMAD1, SMAD2, SMAD3, SMAD4, SMAD6, SMAD7, and SMAD8 at both 2 and 12 weeks; no change in SMAD5 expression was observed in either injury model. The complete gel images are available in Supplementary Figures S1 and S2 for reference. Data are presented as means ± S.D., with a sample size of n = 3 per group (* p < 0.05 vs. Sham group). Abbreviations: wks, weeks.
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    Image Search Results


    Figure 2. Differential expression of SMAD1–8 proteins in the peripheral facial nerve following injury. Western blot analysis of SMAD1–8 protein levels in the peripheral facial nerve at 2 and 12 weeks post injury induced by compression or axotomy. At 2 weeks, compression injury led to a significant increase in SMAD2 expression. By 12 weeks, SMAD7 and SMAD8 expression were elevated in the Compression group. In contrast, axotomy resulted in significant upregulation of SMAD1, SMAD2, SMAD3, SMAD4, SMAD6, SMAD7, and SMAD8 at both 2 and 12 weeks; no change in SMAD5 expression was observed in either injury model. The complete gel images are available in Supplementary Figures S1 and S2 for reference. Data are presented as means ± S.D., with a sample size of n = 3 per group (* p < 0.05 vs. Sham group). Abbreviations: wks, weeks.

    Journal: International journal of molecular sciences

    Article Title: Expression Patterns of SMAD1-8 in the Peripheral Facial Nerve Following Compressive Nerve Injury or Axotomy.

    doi: 10.3390/ijms26052291

    Figure Lengend Snippet: Figure 2. Differential expression of SMAD1–8 proteins in the peripheral facial nerve following injury. Western blot analysis of SMAD1–8 protein levels in the peripheral facial nerve at 2 and 12 weeks post injury induced by compression or axotomy. At 2 weeks, compression injury led to a significant increase in SMAD2 expression. By 12 weeks, SMAD7 and SMAD8 expression were elevated in the Compression group. In contrast, axotomy resulted in significant upregulation of SMAD1, SMAD2, SMAD3, SMAD4, SMAD6, SMAD7, and SMAD8 at both 2 and 12 weeks; no change in SMAD5 expression was observed in either injury model. The complete gel images are available in Supplementary Figures S1 and S2 for reference. Data are presented as means ± S.D., with a sample size of n = 3 per group (* p < 0.05 vs. Sham group). Abbreviations: wks, weeks.

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    Techniques: Quantitative Proteomics, Western Blot, Expressing

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    Article Snippet: Rabbit anti Phospho-Smad1 (Ser463/465)/Smad5 (Ser463/465)/Smad8 (Ser465/467) (D5B10) , Cell Signaling Technology , Cat#11971; RRID:AB_2797785.

    Techniques: Recombinant, Protease Inhibitor, Labeling, Gene Expression, In Situ, Sequencing, Software, Western Blot, Imaging